Paracrine regulation of menstruation

Détails

ID Serval
serval:BIB_F237BEC01709
Type
Article: article d'un périodique ou d'un magazine.
Sous-type
Synthèse (review): revue aussi complète que possible des connaissances sur un sujet, rédigée à partir de l'analyse exhaustive des travaux publiés.
Collection
Publications
Titre
Paracrine regulation of menstruation
Périodique
Journal of Reproductive Immunology
Auteur(s)
Bulletti  C., De Ziegler  D., Albonetti  A., Flamigni  C.
ISSN
0165-0378
Statut éditorial
Publié
Date de publication
08/1998
Peer-reviewed
Oui
Volume
39
Numéro
1-2
Pages
89-104
Notes
Journal Article
Research Support, Non-U.S. Gov't
Review --- Old month value: Aug
Résumé
Endometrial proliferation, secretion, vascular neoformation and modification to shedding is under direct and/or indirect control of steroid hormones. The progressive modification of the endometrial architecture is due to its growth and differentiation. The new tissue regenerates monthly from a 2-5 mm to a 12-18 mm of complex tissue until it sheds under a co-ordinated network of bioactive molecules produced and activated during the menstrual cycle. The steroid hormones, the HLA-DR and integrin molecules, the intense production of several proteins, the vascular damage, and the disconnection of cell-cell and cell-matrix interaction are participating in both the endometrial preparation for embryonic implantation and the shedding and bleeding of the tissue itself. Menstruation is a process associated with damage to the epithelium, endothelium and extracellular matrix, ending on controlled bleeding, tissue dissolution and repair. Endometrial proteinases and tissue factor (TF) contribute to systemic factors to control the mechanisms of regulation of tissue dissolution, tissue shedding, and vascular bleeding during menstruation.
Mots-clé
Animals Apoptosis Endometrium/cytology/physiology Female Gonadal Steroid Hormones/pharmacology Humans *Menstruation Metalloendopeptidases/physiology Uterine Contraction
Pubmed
Web of science
Création de la notice
28/02/2008 12:37
Dernière modification de la notice
03/03/2018 22:40
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