Effectiveness of biomedical risk assessment as an aid for smoking cessation: a systematic review

Details

Serval ID
serval:BIB_EE55099405E1
Type
Article: article from journal or magazin.
Collection
Publications
Institution
Title
Effectiveness of biomedical risk assessment as an aid for smoking cessation: a systematic review
Journal
Tobacco Control
Author(s)
Bize Raphaël, Burnand Bernard, Mueller Yolanda, Cornuz Jacques
ISSN
0964-4563
Publication state
Published
Issued date
2007
Peer-reviewed
Oui
Volume
16
Number
3
Pages
151-156
Language
english
Abstract
OBJECTIVE: To determine the efficacy of biomedical risk assessment (eg, exhaled carbon monoxide (CO), or genetic susceptibility to lung cancer) as an aid for smoking cessation. DATA SOURCES: Cochrane Tobacco Addiction Group Specialized Register, Cochrane Central Register of Controlled Trials, Medline (1966-2004) and EMBASE (1980-2004). STUDY SELECTION: Randomised controlled smoking cessation interventions using biomedical tests with at least 6 months follow-up. DATA EXTRACTION: Two reviewers independently screened all search results (titles and abstracts) for possible inclusion. Each reviewer then extracted data from the selected studies, and assessed their methodological quality based on the CONSORT (Consolidated Standards of Reporting Trials) statement criteria. DATA SYNTHESIS: Of 4049 retrieved references, eight trials were retained for data extraction and analysis. Three trials isolated the effect of exhaled CO on smoking cessation rates resulting in the following ORs and 95% CIs: 0.73 (0.38 to 1.39), 0.93 (0.62 to 1.41) and 1.18 (0.84 to 1.64). Measurement of exhaled CO and spirometry were used together in three trials, resulting in the following ORs (95% CI): 0.60 (0.25 to 1.46), 2.45 (0.73 to 8.25) and 3.50 (0.88 to 13.92). Spirometry results alone were used in one other trial with an OR (95% CI) of 1.21 (0.60 to 2.42). Ultrasonography of carotid and femoral arteries performed on light smokers gave an OR (95% CI) of 3.15 (1.06 to 9.31). CONCLUSIONS: Scarcity and limited quality of the current evidence does not support the hypothesis that biomedical risk assessment increases smoking cessation as compared with the standard treatment.
Keywords
Biofeedback (Psychology) , Smoking , Smoking Cessation
Pubmed
Web of science
Create date
05/02/2008 13:22
Last modification date
20/08/2019 17:15
Usage data