Ramollissements cérébelleux. Présentation clinique et évaluation en tomodensitométrie cérébrale [Cerebellar infarct. Clinical presentation and x-ray computed tomography of the brain]

Détails

ID Serval
serval:BIB_C285DE81D085
Type
Article: article d'un périodique ou d'un magazine.
Collection
Publications
Titre
Ramollissements cérébelleux. Présentation clinique et évaluation en tomodensitométrie cérébrale [Cerebellar infarct. Clinical presentation and x-ray computed tomography of the brain]
Périodique
Schweizerische Medizinische Wochenschrift
Auteur(s)
Uldry P.A., Regli F., Uske A., Bogousslavsky J.
ISSN
0036-7672
Statut éditorial
Publié
Date de publication
1986
Peer-reviewed
Oui
Volume
116
Numéro
2
Pages
34-41
Langue
français
Résumé
A series of 12 patients with cerebellar infarcts diagnosed by computerized tomography are reviewed. The clinical features of cerebellar infarctions cover a wide spectrum, mimicking symptoms and signs from an acute labyrinthitis to a rapidly expanding posterior fossa mass lesion with brain stem and cerebral dysfunction. Two patients were asymptomatic and three showed signs of cerebellar dysfunction only. Three patients had evidence of brain stem dysfunction with cranial nerve palsies accompanying the cerebellar deficit. Two presented a pseudovestibular form with sudden onset of nausea, vomiting, rotary dizziness and ataxia. A pseudotumoral form with intracranial hypertension was found in two cases, in which softening tissue acts as a rapidly expanding posterior foss mass lesion. It is difficult to identify the exact artery involved in a cerebellar infarct because of the collateral circulation and connections between the three major arteries. Atherosclerosis and general decrease in blood flow can be regarded as the most likely factors precipitating focal cerebellar infarction. Surveillance is necessary during the first days with anti-edematous therapy. Rapid deterioration of consciousness should be considered a sign of increasing intracranial pressure progressing with the development of hydrocephalus. If necessary, surgical decompression by external drainage or by direct access to the posterior fossa can be carried out.
Mots-clé
Cerebellar Ataxia/etiology, Cerebellar Diseases/complications, Cerebellar Diseases/etiology, Diagnosis, Differential, Humans, Hypertension/complications, Hypertension/etiology, Tomography, X-Ray Computed
Pubmed
Web of science
Création de la notice
25/07/2008 11:54
Dernière modification de la notice
03/03/2018 21:10
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