Pathogenesis of AF: impact on intracardiac signals.

Détails

ID Serval
serval:BIB_B336A9032381
Type
Article: article d'un périodique ou d'un magazine.
Collection
Publications
Titre
Pathogenesis of AF: impact on intracardiac signals.
Périodique
Conference Proceedings : ... Annual International Conference of the Ieee Engineering In Medicine and Biology Society. Ieee Engineering In Medicine and Biology Society. Conference
Auteur(s)
Shah A.J., Dubois R., Miyazaki S., Jadidi A.S., Scherr D., Wilton S.B., Roten L., Pascale P., Pedersen M., Derval N., Knecht S., Sacher F., Jais P., Narayan S., Hocini M., Haïssaguerre M.
ISSN
1557-170X (Print)
ISSN-L
1557-170X
Statut éditorial
Publié
Date de publication
2011
Volume
2011
Pages
5523-5526
Langue
anglais
Notes
Publication types: Journal ArticlePublication Status: ppublish
Résumé
Atrial fibrillation (AF) is the most common cardiac arrhythmia, and is responsible for the highest number of rhythm-related disorders and cardioembolic strokes worldwide. Intracardiac signal analysis during the onset of paroxysmal AF led to the discovery of pulmonary vein as a triggering source of AF, which has led to the development of pulmonary vein ablation--an established curative therapy for drug-resistant AF. Complex, multicomponent and rapid electrical activity widely involving the atrial substrate characterizes persistent/permanent AF. Widespread nature of the problem and complexity of signals in persistent AF reduce the success rate of ablation therapy. Although signal processing applied to extraction of relevant features from these complex electrograms has helped to improve the efficacy of ablation therapy in persistent/permanent AF, improved understanding of complex signals should help to identify sources of AF and further increase the success rate of ablation therapy.
Mots-clé
Animals, Atrial Fibrillation/physiopathology, Heart Conduction System/physiopathology, Humans, Models, Cardiovascular, Pulmonary Veins/physiopathology
Pubmed
Web of science
Création de la notice
12/06/2014 15:58
Dernière modification de la notice
03/03/2018 20:39
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