Cohort Profile Update: The Swiss Inflammatory Bowel Disease Cohort Study (SIBDCS).
Details
Serval ID
serval:BIB_451608A4AF22
Type
Article: article from journal or magazin.
Collection
Publications
Institution
Title
Cohort Profile Update: The Swiss Inflammatory Bowel Disease Cohort Study (SIBDCS).
Journal
International journal of epidemiology
Contributor(s)
Anderegg C., Bauerfeind P., Beglinger C., Begré S., Belli D., Bengoa J.M., Biedermann L., Bigler B., Binek J., Blattmann M., Boehm S., Borovicka J., Braegger C.P., Brunner N., Bühr P., Burnand B., Burri E., Buyse S., Cremer M., Criblez D.H., de Saussure P., Degen L., Delarive J., Doerig C., Dora B., Dorta G., Egger M., Ehmann T., El-Wafa A., Engelmann M., Ezri J., Felley C., Fliegner M., Fournier N., Fraga M., Frei P., Frei P.R., Fried M., Froehlich F., Funk C., Furlano R.I., Gallot-Lavallée S., Geyer M., Girardin M., Golay D., Grandinetti T., Gysi B., Haack H., Haarer J., Helbling B., Hengstler P., Herzog D., Hess C., Heyland K., Hinterleitner T., Hiroz P., Hirschi C., Hruz P., Iwata R., Jost R., Juillerat P., Keller C., Knellwolf C., Knoblauch C., Köhler H., Koller R., Krieger-Grübel C., Kullak-Ublick G., Künzler P., Landolt M., Lange R., Lehmann F.S., Macpherson A., Maerten P., Maillard M.H., Manser C., Manz M., Marbet U., Marx G., Matter C., Meier R., Mendanova M., Michetti P., Misselwitz B., Morell B., Mosler P., Mottet C., Müller C., Müller P., Müllhaupt B., Münger-Beyeler C., Musso L., Nagy A., Neagu M., Nichita C., Niess J., Nydegger A., Obialo N., Oneta C., Oropesa C., Peter U., Peternac D., Petit L.M., Piccoli-Gfeller F., Pilz J.B., Pittet V., Raschle N., Rentsch R., Restellini R.S., Richterich J.P., Rihs S., Ritz M.A., Roduit J., Rogler D., Rogler G., Rossel J.B., Rueger V., Saner G., Sauter B., Sawatzki M., Schäppi M., Scharl M., Scharl S., Schelling M., Schibli S., Schlauri H., Uebelhart S.S., Schnegg J.F., Schoepfer A., Seibold F., Seirafi M., Semadeni G.M., Semela D., Senning A., Sidler M., Sokollik C., Spalinger J., Spangenberger H., Stadler P., Steuerwald M., Straumann A., Straumann-Funk B., Sulz M., Suter A., Thorens J., Tiedemann S., Tutuian R., Vavricka S., Viani F., Vögtlin J., Von Känel R., Vonlaufen A., Vouillamoz D., Vulliamy R., Wermuth J., Werner H., Wiesel P., Wiest R., Wylie T., Zeitz J., Zimmermann D.
ISSN
1464-3685 (Electronic)
ISSN-L
0300-5771
Publication state
Published
Issued date
01/04/2019
Peer-reviewed
Oui
Volume
48
Number
2
Pages
385-386f
Language
english
Notes
Publication types: Journal Article
Publication Status: ppublish
Publication Status: ppublish
Abstract
The SIBDCS was launched in November 2006 with the aim of enrolling and following patients with inflammatory bowel disease (IBD) throughout Switzerland. Our original sample included incident or prevalent cases, regularly treated in Switzerland, aged 2 and over. Patients were included through their treating gastroenterologist. Data from medical charts and patient self-reported questionnaires were collected annually. Blood samples and biopsies (in case of colonoscopy), collected at baseline and during follow-up, were stored in a biobank. The baseline data collected for the 754 patients recruited in the first 20 months were presented in a previous cohort profile manuscript.
The objectives of the cohort study have been expanded to follow the intense international research activity on IBD. Given the morbidity, unsatisfactory treatments available and their impact on quality of life and costs, much remains to be done to gain more knowledge about IBD pathogenesis and the factors associated with flares or worsening disease. Genetic risk factors alone do not explain the recent increase in disease incidence and are only weakly associated with clinical phenotypes, disease course and severity
The objectives of the cohort study have been expanded to follow the intense international research activity on IBD. Given the morbidity, unsatisfactory treatments available and their impact on quality of life and costs, much remains to be done to gain more knowledge about IBD pathogenesis and the factors associated with flares or worsening disease. Genetic risk factors alone do not explain the recent increase in disease incidence and are only weakly associated with clinical phenotypes, disease course and severity
Pubmed
Web of science
Create date
15/01/2019 11:30
Last modification date
05/11/2021 15:22
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