Expression of FGF-2 in neural progenitor cells enhances their potential for cellular brain repair in the rodent cortex.

Détails

ID Serval
serval:BIB_C3EB0972D9E8
Type
Article: article d'un périodique ou d'un magazine.
Collection
Publications
Titre
Expression of FGF-2 in neural progenitor cells enhances their potential for cellular brain repair in the rodent cortex.
Périodique
Brain
Auteur⸱e⸱s
Dayer A.G., Jenny B., Sauvain M.O., Potter G., Salmon P., Zgraggen E., Kanemitsu M., Gascon E., Sizonenko S., Trono D., Kiss J.Z.
ISSN
1460-2156 (Electronic)
ISSN-L
0006-8950
Statut éditorial
Publié
Date de publication
11/2007
Peer-reviewed
Oui
Volume
130
Numéro
Pt 11
Pages
2962-2976
Langue
anglais
Notes
Publication types: Journal Article ; Research Support, Non-U.S. Gov't
Publication Status: ppublish
Résumé
Strategies to enhance the capacity of grafted stem/progenitors cells to generate multipotential, proliferative and migrating pools of cells in the postnatal brain could be crucial for structural repair after brain damage. We investigated whether the over-expression of basic fibroblast growth factor 2 (FGF-2) in neural progenitor cells (NPCs) could provide a robust source of migrating NPCs for tissue repair in the rat cerebral cortex. Using live imaging we provide direct evidence that FGF-2 over-expression significantly enhances the migratory capacity of grafted NPCs in complex 3D structures, such as cortical slices. Furthermore, we show that the migratory as well as proliferative properties of FGF-2 over-expressing NPCs are maintained after in vivo transplantation. Importantly, after transplantation into a neonatal ischaemic cortex, FGF-2 over-expressing NPCs efficiently invade the injured cortex and generate an increased pool of immature neurons available for brain repair. Differentiation of progenitor cells into immature neurons was correlated with a gradual down-regulation of the FGF-2 transgene. These results reveal an important role for FGF-2 in regulating NPCs functions when interacting with the host tissue and offer a potential strategy to generate a robust source of migrating and immature progenitors for repairing a neonatal ischaemic cortex.
Mots-clé
Animals, Animals, Newborn, Cell Movement, Cell Proliferation, Cerebral Cortex/chemistry, Cerebral Cortex/injuries, Cerebral Cortex/pathology, Fibroblast Growth Factor 2/analysis, Fibroblast Growth Factor 2/genetics, Fibroblast Growth Factor 2/metabolism, Gene Expression, Genetic Engineering, Genetic Vectors/administration & dosage, Genetic Vectors/genetics, HIV-1/genetics, Humans, Hypoxia-Ischemia, Brain/surgery, Immunohistochemistry, Microscopy, Fluorescence, Models, Animal, Rats, Rats, Sprague-Dawley, Stem Cell Transplantation/methods, Stem Cells/metabolism, Stem Cells/pathology, Transduction, Genetic/methods, Transgenes, Wound Healing
Pubmed
Web of science
Open Access
Oui
Création de la notice
19/03/2021 15:44
Dernière modification de la notice
20/03/2021 7:26
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