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Quantitative proteomic analysis reveals AK2 as potential biomarker for late normal tissue radiotoxicity.
10.1186/s13014-019-1351-8
000480487600001
31399108
Lacombe
J.
author
Brengues
M.
author
Mangé
A.
author
Bourgier
C.
author
Gourgou
S.
author
Pèlegrin
A.
author
Ozsahin
M.
author
Solassol
J.
author
Azria
D.
author
article
2019-08-09
Radiation oncology
1748-717X
1748-717X
journal
14
1
142
Adenylate Kinase/metabolism
Biomarkers/metabolism
Breast/metabolism
Breast/radiation effects
Breast Neoplasms/radiotherapy
Female
Fibrosis/etiology
Fibrosis/metabolism
Fibrosis/pathology
Humans
Organs at Risk/radiation effects
Prognosis
Prospective Studies
Proteome/analysis
Radiation Injuries/etiology
Radiation Injuries/metabolism
Radiation Injuries/pathology
Radiation Tolerance
Radiotherapy/adverse effects
Reactive Oxygen Species/metabolism
T-Lymphocytes/metabolism
T-Lymphocytes/pathology
T-Lymphocytes/radiation effects
AK2
NADPH oxidases
Normal tissue radiotoxicity
Proteomics
Radiation-induced breast fibrosis
Radiosensitivity
Radiotherapy
eng
60_published
true
peer-reviewed
Publication types: Journal Article ; Multicenter Study
Publication Status: epublish
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https://serval.unil.ch/resource/serval:BIB_5717C8853286.P001/REF.pdf
http://nbn-resolving.org/urn/resolver.pl?urn=urn:nbn:ch:serval-BIB_5717C88532860
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