La pathogenicite des chlamydiales resistantes aux amibes. [The pathogenicity of amoebae-resisting chlamydiales]

Détails

ID Serval
serval:BIB_F0CF21ECC2A4
Type
Article: article d'un périodique ou d'un magazine.
Sous-type
Synthèse (review): revue aussi complète que possible des connaissances sur un sujet, rédigée à partir de l'analyse exhaustive des travaux publiés.
Collection
Publications
Institution
Titre
La pathogenicite des chlamydiales resistantes aux amibes. [The pathogenicity of amoebae-resisting chlamydiales]
Périodique
Revue Médicale Suisse
Auteur⸱e⸱s
Goy  G., Greub  G.
ISSN
1660-9379 (Print)
Statut éditorial
Publié
Date de publication
08/2005
Volume
1
Numéro
29
Pages
1916-20
Notes
English Abstract
Journal Article
Review --- Old month value: Aug 10
Résumé
During the last 15 years, several new bacterial species related to Chlamydia have been discovered within amoebae. Their ability to grow within these professionnal phagocytes suggests that they are also resistant to destruction by other phagocytic cells such as alveolar macrophages and that they may be excellent candidates as new agents of pneumonia. There are growing evidence that these Chlamydia-like organisms play a role as etiological agent of lower respiratory tract infections. Thus, Parachlamydia is apparently an agent of inhalation pneumonia and--at least in the immunocompromised host--of community-acquired pneumonia, whereas Simkania is considered as a common agent of bronchiolitis and an emerging agent of pneumonia. Nevertheless, their pathogenic potential remains underestimated since these obligate intracellular bacteria grow only poorly or not at all on the culture media generally used to detect pathogen in diagnostic laboratories.
Mots-clé
Amoeba/*microbiology/*pathogenicity Animals Bronchiolitis/etiology/microbiology Chlamydia/*pathogenicity Chlamydia Infections/*microbiology Community-Acquired Infections Humans Immunocompromised Host Pneumonia, Bacterial/*etiology/*microbiology
Pubmed
Création de la notice
25/01/2008 14:28
Dernière modification de la notice
20/08/2019 16:18
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