Hémiataxie et déficit sensitif ipsilatéral. Infarctus du territoire de l'artère choroïdienne antérieure. Diaschisis cérébelleux croisé [Hemiataxia and ipsilateral sensory deficit. Infarct in the area of the anterior choroidal artery. Crossed cerebellar diaschisis]

Détails

ID Serval
serval:BIB_BBD23C824D35
Type
Article: article d'un périodique ou d'un magazine.
Sous-type
Etude de cas (case report): rapporte une observation et la commente brièvement.
Collection
Publications
Titre
Hémiataxie et déficit sensitif ipsilatéral. Infarctus du territoire de l'artère choroïdienne antérieure. Diaschisis cérébelleux croisé [Hemiataxia and ipsilateral sensory deficit. Infarct in the area of the anterior choroidal artery. Crossed cerebellar diaschisis]
Périodique
Revue Neurologique
Auteur(s)
Bogousslavsky J., Regli F., Delaloye B., Delaloye-Bischoff A., Uske A., Despland P.A.
ISSN
0035-3787
Statut éditorial
Publié
Date de publication
1986
Peer-reviewed
Oui
Volume
142
Numéro
8-9
Pages
671-676
Langue
français
Notes
Publication types: Case Reports ; English Abstract ; Journal Article
Résumé
A 58 year old hypertensive man suffered an acute right-sided hemiataxia associated with painful hypoesthesia. CT showed an infarct in the territory of the left anterior choroidal artery. The sensory defect involved all the elementary modes, but predominated on pain sensation. The ataxia suggested a cerebellar type of dysfunction, with hypermetria, intentional tremor, dysdiadochokinesia and a positive rebound phenomenon. There was no hemiparesis, no hemianopia, no neuropsychological dysfunction and no oculomotor abnormality. Hemiataxia with hemisensory defect has not been reported in stroke, and it constitutes a new clinical form of lacunar syndrome. The hemiataxia may be related to the finding on single-photon emission computed tomography (SPECT) using 123 I-amphetamine of a crossed cerebellar diaschisis, which may suggest a transneuronal deactivation. This metabolic depression may have been due to involvement of the temporo-parieto-pontine bundle of Türck in the retro and sub-lenticular portion of the internal capsule. The finding of a normal cortical blood flow may explain why no neuropsychological impairment was present, because of the absence of functional deactivation of the cortex by the underlying deep infarct.
Mots-clé
Arteries, Cerebellar Ataxia/etiology, Cerebellum/blood supply, Cerebral Cortex/blood supply, Cerebral Infarction/diagnosis, Corpus Striatum/blood supply, Humans, Hypesthesia/etiology, Male, Middle Aged, Neural Pathways/blood supply, Pons, Thalamus/blood supply, Tomography, Emission-Computed, Tomography, X-Ray Computed
Pubmed
Web of science
Création de la notice
25/07/2008 10:53
Dernière modification de la notice
20/08/2019 15:29
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