Prestress in the extracellular matrix sensitizes latent TGF-β1 for activation.

Détails

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Etat: Public
Version: Final published version
ID Serval
serval:BIB_A83C9EAA1B9C
Type
Article: article d'un périodique ou d'un magazine.
Collection
Publications
Institution
Titre
Prestress in the extracellular matrix sensitizes latent TGF-β1 for activation.
Périodique
Journal of Cell Biology
Auteur⸱e⸱s
Klingberg F., Chow M.L., Koehler A., Boo S., Buscemi L., Quinn T.M., Costell M., Alman B.A., Genot E., Hinz B.
ISSN
1540-8140 (Electronic)
ISSN-L
0021-9525
Statut éditorial
Publié
Date de publication
2014
Volume
207
Numéro
2
Pages
283-297
Langue
anglais
Notes
Publication types: Journal Article ; Research Support, Non-U.S. Gov't
Publication Status: ppublish
Résumé
Integrin-mediated force application induces a conformational change in latent TGF-β1 that leads to the release of the active form of the growth factor from the extracellular matrix (ECM). Mechanical activation of TGF-β1 is currently understood as an acute process that depends on the contractile force of cells. However, we show that ECM remodeling, preceding the activation step, mechanically primes latent TGF-β1 akin to loading a mechanical spring. Cell-based assays and unique strain devices were used to produce a cell-derived ECM of controlled organization and prestrain. Mechanically conditioned ECM served as a substrate to measure the efficacy of TGF-β1 activation after cell contraction or direct force application using magnetic microbeads. The release of active TGF-β1 was always higher from prestrained ECM as compared with unorganized and/or relaxed ECM. The finding that ECM prestrain regulates the bioavailability of TGF-β1 is important to understand the context of diseases that involve excessive ECM remodeling, such as fibrosis or cancer.
Mots-clé
Animals, Cell Differentiation, Cells, Cultured, Extracellular Matrix/metabolism, HEK293 Cells, Humans, Integrins/metabolism, Integrins/physiology, Mechanotransduction, Cellular, Myofibroblasts/cytology, Myofibroblasts/metabolism, Rats, Wistar, Transforming Growth Factor beta1/metabolism
Pubmed
Web of science
Open Access
Oui
Création de la notice
12/03/2012 12:42
Dernière modification de la notice
20/08/2019 16:12
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