We examined changes in stride temporal parameters and spring-mass model characteristics during repeated sprints following a 3-week period of "live high-train low and high" (LHTLH) altitude training in team-sport players.
While residing under normobaric hypoxia (≥14 h/d; inspired oxygen fraction [FiO2] 14.5%-14.2%) for 14 days, elite field hockey players performed, in addition to their regular field hockey practice in normoxia, 6 sessions (4 × 5 × 5-s maximal sprints; 25-s passive recovery; 5-min rest) under either normobaric hypoxia (LHTLH; FiO2 ∼14.5%, n = 11) or normoxia (live high-train low; FiO2 20.9%, n = 12). A control group (live low-train low; FiO2 ∼20.9%, n = 9) residing in normoxia without additional repeated-sprint training was included. Before (Pre) and a few days (Post-1) and 3 weeks (Post-2) after the intervention, stride mechanics were assessed during an overground repeated-sprint test (8 × 20 m, 20-s recovery). Two-way repeated-measures analysis of variance (time [Pre, Post-1, and Post-2] × condition [LHTLH, live high-train low, and live low-train low]) were conducted.
Peak sprinting speed increased in LHTLH from Pre to Post-1 (+2.2% [2.0%]; P = .002) and Post-2 (+2.0% [2.4%]; P = .025), with no significant changes in live high-train low and live low-train low. There was no main effect of time (all P ≥ .062), condition (all P ≥ .771), or a significant time × condition interaction (all P ≥ .230) for any stride temporal variable (contact time, flight time, stride frequency, and stride length) or spring-mass model characteristics (vertical and leg stiffness).
Peak sprinting speed improved in elite field hockey players following LHTLH altitude training, while stride mechanical adjustments to repeated overground sprints remained unchanged for at least 3 weeks postintervention.